Any structural modification in the tridimensional conformation of the dystrophin (DMD) protein will affect its function because this protein won't be long enough, which causes muscles to rip.
In skeletal muscle and cardiac muscle, dystrophin protein works with other proteins in order to protect muscle fibers from injury as muscle cells contract and relax.
The dystrophin gene is the largest human gene, which encodes for the dystrophin (DMD) protein.
In consequence, different mutations of the DMD gene in people suffering muscular dystrophy disorders have been identified.
The mutations of the DMD gene may cause progressive muscle weakness, muscle atrophy and cardiomyopathies.
In conclusion, any structural modification in the tridimensional conformation of the dystrophin (DMD) protein will affect its function because this protein won't be long enough, which causes muscles to rip.
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