Put the following events of the adaptive immune response in the order they occur.

(a) The antigen-presenting macrophage and some of the bacteria enter the lymph and float down a lymph vessel to the lymph node.

(b) Cytokines, antibodies, and cytotoxic T cells flow through the blood, activating the other immune cells there that are part of the innate immune response and directing them toward the site of infection. The inflammation triggered by the innate immune response allows these immune cells to enter the infected tissue.

(c) You cut yourself and bacteria enter the wound.

(d) At the site of infection, antibodies neutralize, opsonize, and agglutinate the bacteria. The work of the antibodies makes it easier for the phagocytic cells of the innate immune system to destroy the bacteria. Cytotoxic T cells kill body cells that are infected, leaving scar tissue at the site of the wound.

(e) Helper T cells at the lymph node use their T cell receptors to bind the antigens on the surface of the dendrite and bacteria. This triggers the helper T cells to reproduce and to release cytokines.

(f) A dendrite near the cut phagocytizes some bacteria and uses MHC proteins to present exogenous antigens from the destroyed bacteria on its surface.

(g) B cells at the lymph node use their B cell receptors to bind to the antigens on the surface of the dendrite and the bacteria. These B cells also bind to the cytokines being produced. This triggers the B cells to reproduce and to start making lots of antibodies. The cytokines also trigger cytotoxic T cells to reproduce.

The immune response to infection, of whatever kind, is vital for human survival. At its core lies the cells, and enzymes, as well as chemical components, that will activate these responses, the innate, and the acquired immune response.

Although still under lots of study, and science finding new things in this complex system, the order of the steps in which the system is activated, roughly, is this:

1. C. You cut yourself and bacteria enter the wound.

2. F. A dendrite near the cut phagocytizes some bacteria and uses MHC proteins to present exogenous antigens from the destroyed bacteria on its surface

3. A. The antigen-presenting macrophage and some of the bacteria, enter the lymph and float down a lymph vessel to the lymph node, where T cells are awaiting to recognize it and act.

4. E. Helper T cells at the lymph node use their T cell receptors to bind the antigens on the surface of the dendrite and bacteria. This triggers the helper T cells to reproduce, differentiate and release cytokines and other elements that increase inflamation and activate other immune cells.

5. G. Also, B cells at the lymph node use their B cell receptors to bind to the antigens on the surface of the bacteria and dendrite. These B cells also bind to the cytokines being produced. This triggers the B cells to reproduce and start making antibodies. They also trigger cytotoxic T cells CD4 and CD8 differentiation and activation.

6. B. All the elements produced flow through the blood, activating other immune responses and migrate them towards the site of infection. Because of the inflammation, and thanks to the cytokines and protoinflammatory elements, more cells are activated and they can penetrate the inflammed tissue.

7. D. Through the actions of the antibodies, cytokines, chemicals, and different actions of the cells, phagocytes, and cytotoxic T cells are finally able to destroy the invading bacteria and kill all infected tissue as well, leaving behin scar tissue at the site of the wound, and both B and T cells activate their memory capacity, so that if a new infection by that microorganism happens again, the response will be much faster and efficient.

This is a very broad organization of the immune response.